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Augmentation of O-GlcNAcylation via Oga inhibitors, such as Thiamet-G (TMG) or PUGNAc, has been found to exert a negative modulation on synaptic plasticity, enhancing long-term potentiation (LTP) while attenuating long-term depression (LTD) in the hippocampal CA3-CA1 pathway [67]

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