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Large-scale clinical trials have demonstrated that GLP-1RAs reduce the incidence of major adverse cardiovascular events (MACE), including myocardial infarction (MI) and stroke, through metabolic, anti-inflammatory, and direct cardiac mechanisms in both diabetic and non-diabetic populations [316,317,318]

The most noticeable difference in whole-brain activity signatures between GLP-1MK-801 and semaglutide was the significantly higher number of cFOS + cells in the nucleus accumbens in response to GLP-1MK-801 compared with semaglutide and vehicle treatment