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GLP-1RAs have also been shown to attenuate vascular inflammation and oxidative stress, while preserving endothelial barrier integrity and limiting neutrophil infiltration in models of sepsis-associated acute lung injury (ALI) (Wiberg et al., 2018)

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For axon-stimulation experiments, mice expressing ChrimsonR or mCherry in either NTS Gcg terminals in the CeA or CeA Glp1r terminals in the VTA were single-housed for at least 5 days and habituated daily for 1 hour to a patch-cord and for 1 hour to HFD pellets over 2 consecutive days before testing for the hedonic feeding paradigm