Gordan R, Gwathmey JK, Xie LH
Existing evidence is primarily from rodent studies
Contact of nutrients with L-cells in the distal gut likely explains the prolonged duration of elevated GLP-1 during meals, and it might also be responsible for the typically observed increased levels of GLP-1 following gastric bypass and, albeit less so, after sleeve gastrectomy surgery [[211], [212], [213], [214], [215], [216]], because surgical repositioning of the distal gut more rapidly exposes the distal L-cells to incoming nutrients [144]
IV therapy ensures full bioavailability, offering superior results compared to the effects of injections or oral supplementation
Relation between dose of bendroflumethiazide, antihypertensive effect, and adverse biochemical effects