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Renal TECs rely on FAO for energy, with FAOfibrosis crosstalk: FAO blockade promotes lipid deposition during fibrosis, and TGF- reduces FAO to enhance lipid accumulation (204, 205)

Triple agonists take this strategy further by targeting GLP-1R, GIPR, and the glucagon receptor (GCGR), aiming to regulate multiple complementary metabolic pathways

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So it's like chicken, rice and, I don't know, something else, right

It plays a key role in reducing inflammation and oxidative stress in the injured area, further supporting the healing process