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About this article Cite this article Arunim, Dagar, M., Solanki, M

Upon prolonged ER stress, the UPR pathway fails to restore ER homeostasis, thereby the overactivation of UPR shifts toward the apoptotic pathway, where PERK stimulates the pro-apoptotic C/EBP homologous protein (CHOP) expression to initiate ER stress-induced apoptosis (Huang et al

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