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microRNA-449a reduces growth hormone-stimulated senescent cell burden through PI3K-mTOR signaling

In this hypermetabolic state, the body releases a large number of reactive oxygen species (ROS), which can lead to thyroid epithelial cell damage, autoantigen activation of the immune system, and induction of autoimmune deterioration

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Impact of growth hormone replacement therapy on sleep in adult patients with growth hormone deficiency of pituitary origin