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Un estudio de Krivic et al
Studies were eligible if they reported published data in English relevant to endometriosis and at least one of the targeted cell-death modalities, with specific attention to Erastin or its analogs in in-vitro, ex-vivo, in-vivo, or human observational contexts
Figure 7D ), a master transcription factor regulating antioxidant responses and ferroptosis, displays disease state-dependent expression changes reflecting transcriptional adaptive responses of renal cells to ferroptotic stress
Moreover, in an in vitro model of blast-induced auditory hair cell damage, 3-AB inhibition of PARP1 reduced oxidative stress markers, upregulated antioxidant defenses, and helped maintain cell viability by preserving ATP pools 175
[76] Understanding of the pathogenesis of pernicious anaemia increased over subsequent decades
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