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Additionally, hyperhomocysteinemia induces vascular smooth muscle cells (VSMC) proliferation by promoting the expression of adhesion molecules, chemokine and VSMC mitogen leading to several interactions with platelets, clotting factors and lipids [42], and might contribute to the scavenger receptor-mediated uptake of oxidized- Low Density Lipoprotein (LDL) by macrophages, triggering foam cell formation in atherosclerotic plaque [43,44,45,46]

A continuacin se resumen los protocolos de laboratorio habituales los investigadores deben validarlos frente a sus propios SOP internos

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