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In the HK-2 cell model transfected with KCa3.1 siRNA and cultured in a transforming growth factor-beta 1 (TGF-1) environment, as well as in the STZ-induced KCa3.1+/+and KCa3.1-/- mouse models, it was found that calcium-activated potassium channel (KCa3.1) deficiency can restore abnormal mitophagy [120]

10.1016/S0031-9384(97)00261-8 Physiol

Clemastine fumarate had promising clinical effects in a trial of MS patients with chronic optic neuropathy 40,43,44 , possibly also influencing remyelination 41,45

Yang, G., Zhao, K., Ju, Y., Mani, S., Cao, Q., Puukila, S

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