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Long-term safety, carcinogenicity risk, and drug interactions in humans are unknown

B.HegdeM

Mar 2021;53(2):e13953

These results indicate that PNM has potential benefits when used as a filler for conduits in nerve gap injuries

Moreover, NO seems to be the only mediator of cell proliferation arrest through the ERK1/2-p53 signaling pathway ( FIGURE 1 Next to this evidence, in our laboratory it has been demonstrated that GSH depletion modulates the peroxisome proliferator-activated receptor gamma, co-activator 1 alpha (PGC-1) expression and its downstream metabolic pathway in neuronal and skeletal muscle cells ( Figure 1 )