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[8] The same copper that drives the biology is the primary safety liability systemically, and the therapeutic window enough copper to help, not enough to harm is uncharacterized for this route in humans
Over the years, GHK-Cu has been extensively studied in models of skin biology, angiogenesis, and systemic regulation of gene expression
Dietary copper is absorbed in the small intestine at a fractional absorption rate that adjusts inversely to intake : when intake is low, a larger fraction is absorbed
Thus, there is a need for larger studies to fully address this issue
Each cycle accelerates degradation
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