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Researchers also examined major adverse CV events in patient subgroups on the basis of major adverse CV event rates in the placebo group, the presence or absence of CVD, HbA1c level, trial duration, treatment dosing interval, structural homology to human GLP-1 or exendin-4, BMI, age and eGFR

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3 Subsequent work extended these findings to people with type 1 and type 2 diabetes, confirming that amylin-receptor agonism measurably slows emptying across populations

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Pathogenic mutations in IR disrupt these processes and give rise to severe insulin resistance syndromes, including Donohue syndrome, RabsonMendenhall syndrome, and Type A insulin resistance( 612 )