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As expected, oxidative damage and mitochondrial dysfunction are more evident under this condition (Navarro et al., 2008)
4C), thereby supporting the hypothesis that HG-induced EMT contributes to subretinal fibrosis, which represents the end-stage of various eye diseases, including PDR [5]
Subsequently, a postmortem human brain tissue study showed increased NADPH production in the putamen, cortex, and cerebellum of late-stage PD cases, while G6PD was decreased in the putamen of early-stage PD and in the cerebellum of early and late-stage PD
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