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Evidence suggests that IBD pathogenesis is closely linked to gut failure in controlling inflammation, with enteroendocrine cells (EECs) playing a pivotal role in the process (91)

Figure 2 GRP78-specific antibodies were found in up to 63% of RA patients, 7% of patients with other rheumatoid diseases, and only 1% of healthy controls ( In vitro experiments revealed that in synovial cells, TNF and IL-1 increased GRP78 expression

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