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doi: 10.1080/10717544.2016.1177136 113 LemireJCarvalhoKSykesC

This demyelination is both a consequence of N 2 O-induced biochemical disruptions and a key factor in the clinical manifestations of its neurotoxicity

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Additional areas of exploration include its role in -adrenergic receptor signaling within adipose tissue, mechanisms tied to connective tissue and cartilage research, and its noted oral bioactivity in laboratory modelsan uncommon characteristic for small peptides