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Its subsequent metabolism generates ATP, which then inhibits ATP-sensitive potassium channels

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NKp44 + NK cell-mediated destruction of the intestinal epithelium in UC, and HLA-DP401 can engage NKp44 and activate NKp44 + NK cells, mediating damage to intestinal epithelial cells (IECs) in an HLA-DP haplotype-dependent manner

Lab review before starting: A baseline IGF-1, fasting glucose, and (where indicated) a thyroid panel sets the framework for response tracking